POST goosegrass and other grassy weed control in bermudagrass is problematic. Fewer herbicides that can control goosegrass are available due to regulatory pressure and herbicide resistance. Alternative herbicide options that offer effective control are needed. Previous research demonstrates that topramezone controls goosegrass, crabgrass, and other weed species; however, injury to bermudagrass may be unacceptable. The objective of this research was to evaluate the safening potential of topramezone combinations with different additives on bermudagrass. Field trials were conducted at Auburn University during summer and fall from 2015 to 2018 and 2017 to 2018, respectively. Treatments included topramezone mixtures and methylated seed oil applied in combination with five different additives: triclopyr, green turf pigment, green turf paint, ammonium sulfate, and chelated iron. Bermudagrass bleaching and necrosis symptoms were visually rated. Normalized-difference vegetative index measurements and clipping yield data were also collected. Topramezone plus chelated iron, as well as topramezone plus triclopyr, reduced bleaching potential the best; however, the combination of topramezone plus triclopyr resulted in necrosis that outweighed reductions in bleaching. Masking agents such as green turf paint and green turf pigment were ineffective in reducing injury when applied with topramezone. The combination of topramezone plus ammonium sulfate should be avoided because of the high level of necrosis. Topramezone-associated bleaching symptoms were transient and lasted 7 to 14 d on average. Findings from this research suggest that chelated iron added to topramezone and methylated seed oil mixtures acted as a safener on bermudagrass.

Annual applications of three nitrogen (N) rates, two broadleaf herbicides, and six annual-grass herbicides in Kentucky bluegrass (Poa pratensis L.) turf were made from 1974 to 1981. With each increment increase in N (2, 4, and 6 kg N/200 m2), the percentage cover of crabgrass (Digitaria spp.) and number of common dandelion (Taraxacum officinale Weber # TAROF) plants were reduced when compared with the next lower rate. There was an interaction between crabgrass control by the annual-grass herbicides and fertilizer rates. When highly effective herbicides were used, there were no differences in crabgrass control related to N rate. When less effective herbicides or no annual-grass herbicides were used, there were great changes directly related to N rate. Each increment of increased N rate increased the turf quality rating. The broadleaf herbicides resulted in differential quality ratings, although differences were relatively small. Highest quality ratings were for plots treated with the three most effective annual-grass herbicides. Residues in the ppb range of some herbicides were detected in soils sampled about a year after the eighth annual herbicide treatments, but residues were below the phytotoxic level and therefore would have had no effect on control of weeds.

The effects of five preemergence herbicides on turfgrass quality and control of crabgrass (Digitaria spp.) were measured to determine the influence of thatch on preemergence herbicide activity in Kentucky bluegrass (Poa pratensis L.) turf. Field applications of benefin (N-butyl-N-ethyl-α,α,α-trifluoro-2,6-dinitro-p-toluidine), oxadiazon [2-tert-butyl-4-(2,4-dichloro-5-isopropoxyphenyl)-Δ2-1,3,4-oxadiazolin -5-one], and prosulfalin {N-[[4-(dipropylamino)-3,5-dinitrophenyl] sulfonyl]-S,S-dimethylsulfilimine} to thatchy Kentucky bluegrass turf in spring caused injury that was apparent during periods of summer stress, while DCPA (dimethyl tetrachloroterephthalate) and bensulide [O,O-diisopropyl phosphorodithioate S-ester with N-(2-mercaptoethyl)benzenesulfonamide] caused little or no injury. The same treatments on thatch-free sites revealed that only prosulfalin was injurious. Adsorption of benefin, bensulide, and DCPA from solution was greater by thatch than by soil; however, the mobility of these herbicides in undisturbed thatch profiles was greater than measured in undisturbed surface soil from thatch-free sites. Thus, turfgrass injury from preemergence herbicides on thatchy sites is due to at least two factors: the greater mobility of preemergence herbicides in thatch than in soil; and the inherent susceptibility of Kentucky bluegrass to injury from preemergence herbicides that come into direct contact with roots and other absorbing organs.