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This chapter focuses on the perioperative care of the paediatric patient and aims to undermine the common misconception that children are just little adults. Providing safe and effective care for children requires a clear underpinning knowledge of their unique needs. Conscious consideration of age-dependent characteristics such as anatomical, physiological, psychological, and behavioural are essential in the delivery of paediatric patient care. The rationale for adaptations to the delivery of care is to ensure children receive anaesthesia and surgery in a safe and appropriate environment.
The nature and origin of hypoxia is multifaceted. The prevention and treatment include optimisation of pre- and peroxygenation and the wise choice and use of neuromuscular blocking agents. Upper airway reflexes are important to anaesthetists as a clear airway allows safe ventilation of the lungs and oxygenation of the patient. Anaesthetic agents produce changes in the sensitivity of upper airway reflexes, with propofol being associated with depression of upper airway reflexes. This means that airway manipulation, and insertion of airways, including laryngeal mask airways, are more easily tolerated following induction of anaesthesia with propofol compared with other induction agents. Ageing leads to a gradual reduction in the sensitivity of upper airway reflexes. Cigarette smoking increases the sensitivity of upper airway reflexes, a change which persists for up to 2 weeks following cessation of smoking.
Extubation and emergence are high-risk phases of anaesthesia which accounted for 28% of the anaesthesia cases reported to the Fourth National Audit Project of the Difficult Airway Society and the Royal College of Anaesthetists. Problems generally relate to the patient’s anatomy, physiology or to the context in which extubation is carried out. Minor issues such as coughing and breath-holding are common, more serious complications such as aspiration, laryngospasm, post-obstructive pulmonary oedema and hypoxic brain injury are often preventable with proper planning. In this chapter we discuss how to formulate an extubation strategy including risk stratification, planning, awake and deep extubation and modifications aimed at reducing the risk of complications. An awake extubation is suitable for most patients but special techniques such as supraglottic airway exchange, remifentanil infusion or the use of an airway exchange catheter may be helpful in high-risk situations. Post-operative care does not end when the tracheal tube has been removed, handover and documentation are essential components of the extubation plan.
Patients undergoing ear nose and throat (ENT, otorhinolaryngeal) surgery probably present more airway management challenges than any other branch of surgery. ENT procedures encompass a range of operations varying in duration, severity and complexity from simple short cases such as myringotomy, through to complex resection and reconstructive surgeries for head and neck cancer. In all cases the surgical team operates close to the airway and in many within the airway, which is therefore shared with the anaesthetist. In this chapter, the authors discuss in some depth these challenges and how to address them, airway management and ventilation options and strategies including but not limited to awake intubation, different subtypes of jet ventilation, and high flow nasal oxygenation as well recent advances in the field. They further discuss extubation strategies and controversies as well as a plan to manage commonly encountered complications such as bleeding in the airway. For a successful outcome, these ‘shared airway’ procedures require close communication and cooperation between anaesthetist and surgeon, an understanding of each other’s challenges, knowledge of specialist equipment, and a thorough preoperative evaluation to identify potential risk factors for poor perioperative outcomes.
This chapter presents the most common pediatric surgery, myringotomy and ear tube placement. The author reviews in the indications for eat tubes in the setting of a child with upper respiratory tract infection. The perioperative considerations for upper respiratory tract infection are considered with relation to case postponement.
The incidence of difficult airway is higher in patients undergoing ENT surgery and, specifically, in patients undergoing ENT cancer surgery. Even the process of topicalization with local anesthetic can precipitate loss of the airway, as can some of the complications associated with awake intubation (e.g. airway bleeding and laryngospasm). The preoperative interview should also address the possibility of events having occurred since the last anesthetic such as weight gain, laryngeal stenosis from previous airway intervention, airway radiation, facial cosmetic surgery, and worsening temporomandibular joint disorder or rheumatoid arthritis. Prior to awake intubation, premedication is commonly used to reduce secretions, enable adequate topicalization of the airway, reduce the risk of epistaxis, and protect against the risk of aspiration. Depending on the clinical circumstance, intravenous sedation may be useful in allowing the patient to tolerate awake intubation by providing anxiolysis, amnesia, and analgesia.
An increase in the sensitivity of airway reflexes during induction of anaesthesia increases the likelihood of laryngeal spasm and coughing. Some early work identified two types of receptor in the larynx: one a slowly adapting receptor and the second a rapidly adapting receptor thought to be especially sensitive to chemical stimulants. Anaesthetic agents may sensitise the receptors, explaining why some inhaled and intravenous agents may easily precipitate laryngeal spasm. Prior to the administration of lidocaine airway irritation caused not only the cough reflex, but also other respiratory reflexes such as expiration, apnoea and spasmodic panting. It should be noted that the initial application of local anaesthetic agents to the airway may be associated with laryngospasm. It is now thought that the pharyngeal dilators, in addition to the diaphragm, comprise the efferent output of the respiratory centre. Tonic contraction is required to keep the tongue forward and maintain airway patency.
An extubation plan should always be formulated. Extubation in a deep plane of anaesthesia is an advanced technique. One-third of aspiration events occur after extubation. Every extubation technique should ensure minimal interruption in the delivery of oxygen to the patient's lungs, and should extubation fail, ventilation should be achievable with the minimal difficulty or delay. The choice of extubation position reflects a balance between the risks of vomiting post-extubation, and subsequent inhalation and soiling of the lungs, and potential respiratory embarrassment and ease of assisting ventilation. The depth of anaesthesia at the time of extubation is highly important because of the risk of life-threatening laryngospasm. Peri-extubation insertion of a laryngeal mask airway (LMA) is a useful technique for airway maintenance in the recovery period with less airway obstruction and coughing, and higher saturations than either deep or awake extubation. An airway exchange catheter (AEC) is a useful aid.
(1) To present a rare case of stridor secondary to prolonged laryngospasm in a patient with Parkinson's disease, and (2) to review the literature on stridor in Parkinson's disease.
Methods:
We report a 73-year-old Parkinson's disease patient who developed acute stridor due to prolonged laryngospasm triggered by overspill of excessive secretions. The literature was reviewed, following a Medline search using the keywords ‘Parkinson's disease’ and ‘stridor’ or ‘airway obstruction’ or ‘laryngospasm’ or ‘laryngeal dystonia’ or ‘bilateral vocal cord palsy’.
Result:
Only 12 previously reported cases of stridor in Parkinson's disease patients were identified. Causes included bilateral vocal fold palsy (eight cases), laryngospasm (five), and dystonia of the jaw and neck muscles (two). The mechanism of laryngospasm in our patient was similar to ‘dry drowning’, and has not previously been described.
Conclusion:
Laryngospasm can be triggered in Parkinson's disease by excessive secretions entering the larynx. The mechanism is similar to ‘dry drowning’. Treatment focuses on reducing secretions. The use of botulinum toxin to reduce spasm is inappropriate in this situation. This case emphasises the importance of recognising different causes of stridor in Parkinson's disease patients, as this affects management.
To review the presentation, risk factors and management of paroxysmal laryngospasm.
Study design:
Retrospective review of cases.
Setting:
A teaching hospital otolaryngology department with a subspecialty interest in airway disorders.
Patients:
All patients diagnosed with laryngospasm over a two-year period were reviewed. Information was obtained about disease presentation, risk factors, management and symptom resolution.
Results:
Laryngospasm was diagnosed in nine women and six men. The average age at presentation was 56±6.5 years, and there was an 80 per cent association with gastroesophageal reflux disease. Proton pump inhibitors led to complete symptom resolution in six patients and to partial symptomatic relief, requiring no further treatment, in a further four patients. Of the remaining five patients unresponsive to proton pump inhibitor therapy, two continued to experience syncopal episodes due to laryngospasm. Both these patients achieved complete remission after laryngeal botulinum toxin injection. Symptoms recurred after three to four months and were successfully treated with a repeat injection.
Conclusions:
The primary risk factor for spontaneous laryngospasm is laryngopharyngeal reflux. Symptoms are distressing and may be relieved in most cases by treatment aimed at suppressing gastric acid secretion. Laryngeal botulinum toxin injection appears to be a viable treatment modality in selected patients with refractory symptoms.
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