The asymptotic behaviour of many locally branching epidemic models can, at least to first order, be deduced from the limit theory of two branching processes. The first is Whittle's (1955) branching approximation to the early stages of the epidemic, the phase in which approximately exponential growth takes place. The second is the susceptibility approximation; the backward branching process that approximates the history of the contacts that would lead to an individual becoming infected. The simplest coupling arguments for demonstrating the closeness of these branching process approximations do not keep the processes identical for quite long enough. Thus, arguments showing that the differences are unimportant are also needed. In this paper we show that, for some models, couplings can be constructed that are sufficiently accurate for this extra step to be dispensed with.

We consider a stochastic SIR (susceptible → infective → removed) epidemic on a random graph with specified degree distribution, constructed using the configuration model, and investigate the ‘acquaintance vaccination’ method for targeting individuals of high degree for vaccination. Branching process approximations are developed which yield a post-vaccination threshold parameter, and the asymptotic (large population) probability and final size of a major outbreak. We find that introducing an imperfect vaccine response into the present model for acquaintance vaccination leads to sibling dependence in the approximating branching processes, which may then require infinite type spaces for their analysis and are generally not amenable to numerical calculation. Thus, we propose and analyse an alternative model for acquaintance vaccination, which avoids these difficulties. The theory is illustrated by a brief numerical study, which suggests that the two models for acquaintance vaccination yield quantitatively very similar disease properties.

In this paper we characterise the distributions of the number of predecessors and of the number of successors of a given set of vertices, A, in the random mapping model, TnD̂ (see Hansen and Jaworski (2008)), with exchangeable in-degree sequence (D̂1,D̂2,…,D̂n). We show that the exact formulae for these distributions and their expected values can be given in terms of the distributions of simple functions of the in-degree variables D̂1,D̂2,…,D̂n. As an application of these results, we consider two special examples of TnD̂ which correspond to random mappings with preferential and anti-preferential attachment, and determine the exact distributions for the number of predecessors and the number of successors in these cases. We also characterise, for these two special examples, the asymptotic behaviour of the expected numbers of predecessors and successors and interpret these results in terms of the threshold behaviour of epidemic processes on random mapping graphs. The families of discrete distributions obtained in this paper are also of independent interest.

In this paper we consider a stochastic SIR (susceptible→infective→removed) epidemic model in which individuals may make infectious contacts in two ways, both within ‘households’ (which for ease of exposition are assumed to have equal size) and along the edges of a random graph describing additional social contacts. Heuristically motivated branching process approximations are described, which lead to a threshold parameter for the model and methods for calculating the probability of a major outbreak, given few initial infectives, and the expected proportion of the population who are ultimately infected by such a major outbreak. These approximate results are shown to be exact as the number of households tends to infinity by proving associated limit theorems. Moreover, simulation studies indicate that these asymptotic results provide good approximations for modestly sized finite populations. The extension to unequal-sized households is discussed briefly.

This paper is concerned with a nonstationary Markovian chain of cascading damage that constitutes an iterated version of a classical damage model. The main problem under study is to determine the exact distribution of the total outcome of this process when the cascade of damages finally stops. Two different applications are discussed, namely the final size for a wide class of SIR (susceptible → infective → removed) epidemic models and the total number of failures for a system of components in reliability. The starting point of our analysis is the recent work of Lefèvre (2007) on a first-crossing problem for the cumulated partial sums of independent parametric distributions, possibly nonstationary but stable by convolution. A key mathematical tool is provided by a nonstandard family of remarkable polynomials, called the generalised Abel–Gontcharoff polynomials. Somewhat surprisingly, the approach followed will allow us to relax some model assumptions usually made in epidemic theory and reliability. To close, approximation by a branching process is also investigated to a certain extent.

We consider a stochastic model for the spread of an SIR (susceptible → infective → removed) epidemic among a closed, finite population that contains several types of individuals and is partitioned into households. The infection rate between two individuals depends on the types of the transmitting and receiving individuals and also on whether the infection is local (i.e., within a household) or global (i.e., between households). The exact distribution of the final outcome of the epidemic is outlined. A branching process approximation for the early stages of the epidemic is described and made fully rigorous, by considering a sequence of epidemics in which the number of households tends to infinity and using a coupling argument. This leads to a threshold theorem for the epidemic model. A central limit theorem for the final outcome of epidemics which take off is derived, by exploiting an embedding representation.

The time until extinction for the closed SIS stochastic logistic epidemic model is investigated. We derive the asymptotic distribution for the extinction time as the population grows to infinity, under different initial conditions and for different values of the infection rate.

We study the long-term behaviour of a sequence of multitype general stochastic epidemics, converging in probability to a deterministic spatial epidemic model, proposed by D. G. Kendall. More precisely, we use branching and deterministic approximations in order to study the asymptotic behaviour of the total size of the epidemics as the number of types and the number of individuals of each type both grow to infinity.

The paper is first concerned with a comparison of the partial sums associated with two sequences of n exchangeable Bernoulli random variables. It then considers a situation where such partial sums are obtained through an iterative procedure of branching type stopped at the first-passage time in a linearly decreasing upper barrier. These comparison results are illustrated with applications to certain urn models, sampling schemes and epidemic processes. A key tool is a non-standard hierarchical class of stochastic orderings between discrete random variables valued in {0, 1,· ··, n}.

This paper describes the early and final properties of a general S–I–R epidemic process in which the infectives behave independently, each infective has a random number of contacts with the others in the population, and individuals vary in their susceptibility to infection. For the case of a large initial number of susceptibles and a small (finite) initial number of infectives, we derive the threshold behavior and the limiting distribution for the final state of the epidemic. Also, we show strong convergence of the epidemic process over any finite time interval to a birth and death process, extending the results of Ball (1983). These complement some results due to Butler (1994), who considers the case of a large initial number of infectives.

By imbedding the multitype version of the standard epidemic model in a multiparameter process, we derive a functional limit theorem for the total cost of the epidemics.

A generalization of Downton's (1968) carrier-borne epidemic process is considered in which the assumption of homogeneous mixing is replaced by a general infection mechanism. Using a method and notation similar to that of Gani and Purdue (1984), a recursive algorithm for the joint distribution of the total size and the total observed size of the epidemic is obtained.

A random mapping of a countable set N into itself is studied. An explicit formula is found for the probability that a subset of N will not contain cycles of the mapping. This formula is applied then for a detailed asymptotic analysis of two special mappings.

A random graph is a collection of n points and n directed arcs: a directed arc goes equiprobably from each point to one of (n – 1) other points. m points are initially ‘infected'. We consider several schemes of epidemic process, e.g. when the infection is delivered according to arc direction. We prove that the probability of infecting all the n points with m = 1 is ∼ e/n, when n → ∞; another result is that m = o(√ n) cannot infect an essential part of the graph (having the size of O(n)). Possible applications of the models to real world phenomena are briefly discussed.