Skip to main content Accessibility help

We use cookies to distinguish you from other users and to provide you with a better experience on our websites. Close this message to accept cookies or find out how to manage your cookie settings.

Close cookie message
×
  1. Home
  2. Search

Refine search

Refine search

Access:
Content type:
Publication date:
Apply   From and To filters
Subject: Show more
Journals Show more
Publishers: Show more
Societies Show more
Series: Show more
Collections: Show more

Actions for selected content:

Select all | Deselect all
  • View selected items
  • Export citations
  • Download PDF (zip)
  • Save to Kindle
  • Save to Dropbox
  • Save to Google Drive

Save Search

You can save your searches here and later view and run them again in "My saved searches".

Please provide a title, maximum of 40 characters.
Cancel
×

1 results

  • Case 96 - Guillain-Barrésyndrome

  • from Section II - Neurocritical care
  • Edited by George A. Mashour, Ehab Farag
  • Book:
    Case Studies in Neuroanesthesia and Neurocritical Care
    Published online:
    03 May 2011
    Print publication:
    03 February 2011, pp 324-326

  • Summary

    Subarachnoid hemorrhage (SAH) represents bleeding into the subarachnoid space, most commonly from a ruptured cerebral aneurysm. This chapter presents a case study of a 52-year-old right-handed female with a medical history of smoking and hypertension who developed an abrupt onset of a severe bifrontal headache. The development of the operative microscope and interventional procedures has allowed for the early treatment of cerebral aneurysms to prevent rebleeding. Rebleeding occurs within the first few weeks of the initial hemorrhage in one-third of patients with SAH and acutely worsens outcomes. Treatment of patients with SAH, once an aneurysm is secured, includes generous use of intravenous isotonic fluids. Clinical studies have suggested that a majority of neurologic deficits can be reversed with the application of the hemodynamic techniques. The application of interventions for vasospasm may be guided by noninvasive measures of vessel narrowing and cerebral blood flow.