Hostname: page-component-78c5997874-lj6df Total loading time: 0 Render date: 2024-11-14T22:11:25.505Z Has data issue: false hasContentIssue false
  • English
  • Français

Posterior Cuts in Prefrontal Leucotomy: A Clinico-Pathological Study

Published online by Cambridge University Press:  08 February 2018

Alfred Meyer  and
Turner McLardy
Alfred Meyer
Affiliation:
Department of Neuropathology, Research Laboratory, Maudsley Hospital, London
Turner McLardy
Affiliation:
Department of Neuropathology, Research Laboratory, Maudsley Hospital, London
Get access Rights & Permissions [Opens in a new window]

Extract

The usual leucotomy cut in this country, made through burr holes orientated by means of skull measurements, in the majority of cases enters the brain at about the middle of pars triangularis of the inferior frontal gyrus (Brodmann's Area 45), and if performed in a strictly coronal plane remains entirely within the prefrontal region. Intentionally or unintentionally cuts are not infrequently placed either anterior or posterior to this plane. That cuts made posterior to strictly prefrontal levels are often followed by an unfavourable post-operative course distinguished by particularly severe personality change, persisting incontinence, vasomotor, trophic and neurological disturbances has been demonstrated by Knight (1943), Meyer and Beck (1945), Ziegler and Osgood (1945), Reitman (1946) and others.

Type
Part I.—Original Articles
Information
Journal of Mental Science , Volume 94 , Issue 396 , July 1948 , pp. 555 - 564
Copyright
Copyright © Royal College of Psychiatrists, 1948 

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

Bailey, P., and Bremer, F. (1938), J. Neurophysiol., 1, 405.CrossRefGoogle Scholar
Bailey, P., and Sweet, W. H. (1940), ibid., 3, 276.Google Scholar
Cumings, J. N. (1942), J. Neurol. Psychiat., 5, 40.CrossRefGoogle Scholar
Delgado, J. M. R., Fulton, J. F., and Livingstone, R. B. (1947), Fed. Proc., 6, 96.Google Scholar
Fulton, J. F. (1947), Acta Med. Scand., Suppl., 196, 617.CrossRefGoogle Scholar
Idem , Livingstone, R. B., and Davis, G. D. (1947), Fed. Proc., 6, 108.Google Scholar
Heath, R. G., Freedman, D. A., and Mettler, F. A. (1947), ibid., 6, 126.Google Scholar
Hebb, D. O. (1945), Arch. Neurol. Psychiat., 54, 10.CrossRefGoogle Scholar
Johnson, H. C., Walker, A. E., Case, T. J., and Kollross, J. J. (1946), ibid., 56, 184.Google Scholar
Kennard, M. A. (1935), ibid., 33, 537.Google Scholar
Knight, G. C. (1943), J. Ment. Sci., 89, 174.Google Scholar
Kremer, W. F. (1947), J. Neurophysiol., 10, 371.CrossRefGoogle Scholar
McCulloch, W. S. (1944), Physiol. Rev., 24, 390.CrossRefGoogle Scholar
Mettler, F. A. (1945), J. Neuropath. Exper. Neurol., 4, 99.CrossRefGoogle Scholar
Meyer, A., and Beck, E. (1945), J. Ment. Sci., 91, 411.CrossRefGoogle Scholar
Meyer, A., and McLardy, T. (1947), ibid., 90, 7.Google Scholar
Meyer, A., Beck, E., and McLardy, T. (1947), Brain, 70, 18.CrossRefGoogle Scholar
Meyer, A., McLardy, T., and Beck, E. (1948, in press), Psychiat. en Neurol. Bladen. Google Scholar
Pennybacker, J. (1945), Proc. Roy. Soc. Med., 38, 431.Google Scholar
Reitman, F. (1946), Brit. Med. J., 1, 235.CrossRefGoogle Scholar
Richter, C. P., and Hines, M. (1938), Brain, 61, 1.CrossRefGoogle Scholar
Ruch, T. C., and Shenkin, H. A. (1943), J. Neurophysiol., 6, 349.CrossRefGoogle Scholar
Russell, W. R. (1947), Brain, 70, 225.CrossRefGoogle Scholar
Smith, W. K. (1938), J. Neurophysiol., 1, 55.CrossRefGoogle Scholar
Idem (1945), ibid., 8, 241.Google Scholar
Walker, A. E. (1940), J. Comp. Neurol., 73, 59.CrossRefGoogle Scholar
Ziegler, L. H., and Osgood, C. W. (1945), Arch. Neurol. Psychiat., 53, 262.CrossRefGoogle Scholar
Submit a response

eLetters

No eLetters have been published for this article.