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While chapter 3 outlines how colonial indirect rule created the structural conditions for Maoist insurgency through multiple causal pathways, it also emphasizes that rebel agency in the form of ideological frames and organizational legacies plays an equally important role in mobilizing the ethnic networks of lower castes and tribes. In this chapter, I provide qualitative evidence to demonstrate this interaction between rebel agency and structural conditions created by colonial institutions. I first provide a detailed history of the organizational evolution of the various Maoist factions since the 1960s. Then, I use fieldwork interviews and textual analysis of Maoist documents to demonstrate that the Maoists are strategic about their choice of area, based on local politics, terrain, ethnic composition, and level of economic development. However, even within those areas they considered favorable based on terrain, inequality, and politics, they were successful only where the British colonial institutions of zamindari (landlord) land tenure or certain types of princely states were present. This demonstrates that rebel agency interacts with and is constrained by the opportunity structures of land/ethnic inequality available in areas of former indirect rule and revenue collection.
This final chapter draws together the complex claims made in the previous chapters into a single overall argument setting out the causal pathway to the genocide. It then takes stock of what we have cumulatively learned about Rwanda’s genocide in the twenty-five years that have elapsed since it occurred and explains the ways in which this book either reinforces or extends the scholarly consensus. It also articulates the book’s more novel contributions to our knowledge and understanding of Rwanda’s genocide and other cases. The chapter finally concludes by considering the broader theoretical implications that the book’s findings have for genocides and mass killings more generally.
Preterm birth is associated with an increased risk for cognitive-neurophysiological impairments and attention-deficit/hyperactivity disorder (ADHD). Whether the associations are due to the preterm birth insult per se, or due to other risk factors that characterise families with preterm-born children, is largely unknown.
Methods
We employed a within-sibling comparison design, using cognitive-performance and event-related potential (ERP) measures from 104 preterm-born adolescents and 104 of their term-born siblings. Analyses focused on ADHD symptoms and cognitive and ERP measures from a cued continuous performance test, an arrow flanker task and a reaction time task.
Results
Within-sibling analyses showed that preterm birth was significantly associated with increased ADHD symptoms (β = 0.32, p = 0.01, 95% CI 0.05 to 0.58) and specific cognitive-ERP impairments, such as IQ (β = −0.20, p = 0.02, 95% CI −0.40 to −0.01), preparation-vigilance measures and measures of error processing (ranging from β = 0.71, −0.35). There was a negligible within-sibling association between preterm birth with executive control measures of inhibition (NoGo-P3, β = −0.07, p = 0.45, 95% CI −0.33 to 0.15) or verbal working memory (digit span backward, β = −0.05, p = 0.63, 95% CI −0.30 to 0.18).
Conclusions
Our results suggest that the relationship between preterm birth with ADHD symptoms and specific cognitive-neurophysiological impairments (IQ, preparation-vigilance and error processing) is independent of family-level risk and consistent with a causal inference. In contrast, our results suggest that previously observed associations between preterm birth with executive control processes of inhibition and working memory are instead linked to background characteristics of families with a preterm-born child rather than preterm birth insult per se. These findings suggest that interventions need to target both preterm-birth specific and family-level risk factors.
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