We introduce an extension to Kermack and McKendrick’s classic susceptible–infected–recovered (SIR) model in epidemiology, whose underlying mechanism of infection consists of individuals attending randomly generated social gatherings. This gives rise to a system of ordinary differential equations (ODEs) where the force of the infection term depends non-linearly on the proportion of infected individuals. Some specific instances yield models already studied in the literature, to which the present work provides a probabilistic foundation. The basic reproduction number is seen to depend quadratically on the average size of the gatherings, which may be helpful in understanding how restrictions on social gatherings affect the spread of the disease. We rigorously justify our model by showing that the system of ODEs is the mean-field limit of the jump Markov process corresponding to the evolution of the disease in a finite population.

We explore a simple model of network dynamics which has previously been applied to the study of information flow in the context of epidemic spreading. A random rooted network is constructed that evolves according to the following rule: at a constant rate, pairs of nodes (i, j) are randomly chosen to interact, with an edge drawn from i to j (and any other out-edge from i deleted) if j is strictly closer to the root with respect to graph distance. We characterise the dynamics of this random network in the limit of large size, showing that it instantaneously forms a tree with long branches that immediately collapse to depth two, then it slowly rearranges itself to a star-like configuration. This curious behaviour has consequences for the study of the epidemic models in which this information network was first proposed.

During an epidemic outbreak, typically only partial information about the outbreak is known. A common scenario is that the infection times of individuals are unknown, but individuals, on displaying symptoms, are identified as infectious and removed from the population. We study the distribution of the number of infectives given only the times of removals in a Markovian susceptible–infectious–removed (SIR) epidemic. Primary interest is in the initial stages of the epidemic process, where a branching (birth–death) process approximation is applicable. We show that the number of individuals alive in a time-inhomogeneous birth–death process at time $t \geq 0$, given only death times up to and including time t, is a mixture of negative binomial distributions, with the number of mixing components depending on the total number of deaths, and the mixing weights depending upon the inter-arrival times of the deaths. We further consider the extension to the case where some deaths are unobserved. We also discuss the application of the results to control measures and statistical inference.

We determine the distributions of some random variables related to a simple model of an epidemic with contact tracing and cluster isolation. This enables us to apply general limit theorems for super-critical Crump–Mode–Jagers branching processes. Notably, we compute explicitly the asymptotic proportion of isolated clusters with a given size amongst all isolated clusters, conditionally on survival of the epidemic. Somewhat surprisingly, the latter differs from the distribution of the size of a typical cluster at the time of its detection, and we explain the reasons behind this seeming paradox.

Across a wide variety of applications, the self-exciting Hawkes process has been used to model phenomena in which the history of events influences future occurrences. However, there may be many situations in which the past events only influence the future as long as they remain active. For example, a person spreads a contagious disease only as long as they are contagious. In this paper, we define a novel generalization of the Hawkes process that we call the ephemerally self-exciting process. In this new stochastic process, the excitement from one arrival lasts for a randomly drawn activity duration, hence the ephemerality. Our study includes exploration of the process itself as well as connections to well-known stochastic models such as branching processes, random walks, epidemics, preferential attachment, and Bayesian mixture models. Furthermore, we prove a batch scaling construction of general, marked Hawkes processes from a general ephemerally self-exciting model, and this novel limit theorem both provides insight into the Hawkes process and motivates the model contained herein as an attractive self-exciting process in its own right.

We couple a multi-type stochastic epidemic process with a directed random graph, where edges have random weights (traversal times). This random graph representation is used to characterise the fractions of individuals infected by the different types of vertices among all infected individuals in the large population limit. For this characterisation, we rely on the theory of multi-type real-time branching processes. We identify a special case of the two-type model in which the fraction of individuals of a certain type infected by individuals of the same type is maximised among all two-type epidemics approximated by branching processes with the same mean offspring matrix.

In this paper we aim to apply simple actuarial methods to build an insurance plan protecting against an epidemic risk in a population. The studied model is an extended SIR epidemic in which the removal and infection rates may depend on the number of registered removals. The costs due to the epidemic are measured through the expected epidemic size and infectivity time. The premiums received during the epidemic outbreak are measured through the expected susceptibility time. Using martingale arguments, a method by recursion is developed to calculate the cost components and the corresponding premium levels in this extended epidemic model. Some numerical examples illustrate the effect of removals and the premium calculation in an insurance plan.

We prove that, for Poisson transmission and recovery processes, the classic susceptible→infected→recovered (SIR) epidemic model of Kermack and McKendrick provides, for any given time t>0, a strict lower bound on the expected number of susceptibles and a strict upper bound on the expected number of recoveries in the general stochastic SIR epidemic. The proof is based on the recent message passing representation of SIR epidemics applied to a complete graph.

The asymptotic behaviour of many locally branching epidemic models can, at least to first order, be deduced from the limit theory of two branching processes. The first is Whittle's (1955) branching approximation to the early stages of the epidemic, the phase in which approximately exponential growth takes place. The second is the susceptibility approximation; the backward branching process that approximates the history of the contacts that would lead to an individual becoming infected. The simplest coupling arguments for demonstrating the closeness of these branching process approximations do not keep the processes identical for quite long enough. Thus, arguments showing that the differences are unimportant are also needed. In this paper we show that, for some models, couplings can be constructed that are sufficiently accurate for this extra step to be dispensed with.

Motivated by the analysis of social networks, we study a model of random networks that has both a given degree distribution and a tunable clustering coefficient. We consider two types of growth process on these graphs that model the spread of new ideas, technologies, viruses, or worms: the diffusion model and the symmetric threshold model. For both models, we characterize conditions under which global cascades are possible and compute their size explicitly, as a function of the degree distribution and the clustering coefficient. Our results are applied to regular or power-law graphs with exponential cutoff and shed new light on the impact of clustering.