Attention-deficit/hyperactivity disorder (ADHD) is a neurodevelopmental disorder characterized by inattention and/or hyperactivity-impulsivity, accompanied by deficits in executive function (EF). However, how the two core symptoms of ADHD are affected by EF deficits remains unclear. 649 children with ADHD were recruited. Data were collected from ADHD rating scales, the Behavior Rating Inventory of EF (BRIEF), and other demographic questionnaires. Regression and path analyses were conducted to explore how deficits in cool and hot EF influence different ADHD core symptoms. Latent class analysis and logistic regression were employed to further examine whether classification of ADHD subtypes is associated with specific EF deficits. EF deficits significantly predicted the severity of ADHD core symptoms, with cool EF being a greater predictor of inattention and hot EF having a more significant effect on hyperactivity/impulsivity. Moreover, person-centered analyses revealed higher EF deficits in subtypes of ADHD with more severe symptoms, and both cool and hot EF deficits could predict the classification of ADHD subtypes. Our findings identify distinct roles for cool and hot EF deficits in the two core symptoms of ADHD, which provide scientific support for the development of ADHD diagnostic tools and personalized intervention from the perspective of specific EF deficits.

The goal of this study was to unpack processes that may lead to child emotional insecurity. Guided by the emotional security theory (EST/EST-R), we examined the mediational role of parental depressive symptomology between interparental conflict (IPC), both constructive and destructive, and child emotional insecurity at age 36-months. We partitioned unique variance of IPC from shared using an extension of the common fate model. We used two-wave data from the Building Strong Families project, which consisted of racially diverse couples/parents (N = 4,424) who were low income and unmarried at the conception of their child. We found gendered differences for how mothers and fathers experience IPC, with mothers more influenced by their relational circumstances. We also found that fathers were vulnerable to experiencing depressive symptoms following aspects of destructive IPC. Consistent with EST-R, constructive IPC did not promote emotional security in children. Rather, both destructive and constructive IPC related to greater levels of emotional insecurity, with destructive IPC showing stronger effects. Proposed mediation was found for fathers only. Our findings may appeal to scholars who focus on untangling the complexity of IPC and intervention specialists and clinicians interested in a process-oriented approaches to the development of child psychopathology.

The association between low household income and adolescent mental health causes continuing concern. We examined the relation between household income and adolescent internalizing and externalizing problems, and explored individual, parental, and neighborhood characteristics. The sample included 872 Dutch adolescents (Mage = 14.93 years) oversampled on risk of psychopathology. Low income was defined as parent-reported net monthly household income below the 20th percentile (<€2000). Internalizing and externalizing problems were examined using the Youth Self-Report and Child Behavior Checklist. Covariates included sex, age, ethnic background, IQ, perceived social support, adverse life events, physical health, parental psychopathology, parental IQ, parent-child interaction, neighborhood unemployment rate, and neighborhood violence. Low household income was associated with more internalizing and externalizing problems. These associations were explained by more physical health concerns, increased parental psychopathology, more parent-child interaction problems, more adverse life events, lower perceived social support, and lower adolescent IQ. For all, except for mother-child interaction, a mediating role was suggested. This indicates a complex interplay between household income, individual, social, and parental factors affecting adolescent mental health. This study accentuates the necessity for a comprehensive, multi-faceted approach to address the negative effects of poverty on adolescent mental health, targeting these influences for preventive measures.

Children adopted from public care are more likely to experience mental health problems associated with their histories of early life adversity and instability, but few studies have investigated children’s mental health longitudinally across developmental turning points. We followed a sample of N = 92 UK domestically adopted children and their families at six time points over eight years post-placement (children’s ages ranged from 2 to 15 years). We used multilevel growth curve analysis to model time-related changes in children’s internalizing symptoms and externalizing problems and spline models to investigate patterns of change before and after school entry. Children’s internalizing symptoms followed a linear increasing trajectory, and externalizing problems followed a quadradic pattern where problems accelerated in early childhood and decelerated in late childhood. Spline models indicated an elevation in internalizing symptoms and externalizing problems as children started school. Internalizing symptoms continued to increase over time after school started and externalizing problems plateaued after the first years in school. Children adopted close in time to school entry displayed more problems when they started school. The transition to school represents a time of vulnerability for adopted children, especially for those who are adopted close in time to this transition, underscoring the need for ongoing support for their mental health across childhood.

The role of the gut microbiome in infant development has gained increasing interest in recent years. Most research on this topic has focused on the first three to four years of life because this is a critical period for developing gut-brain connections. Prior studies have identified associations between the composition and diversity of the gut microbiome in infancy and markers of temperament, including negative affect. However, the specific microbes affected, and the directionality of these associations have differed between studies, likely due to differences in the developmental period of focus and assessment approaches. In the current preregistered study, we examined connections between the gut microbiome, assessed at two time points in infancy (2 weeks and 18 months), and negative affect measured at 30 months of age in a longitudinal study of infants and their caregivers. We found that infants with higher gut microbiome diversity at 2 weeks showed more observed negative affect during a study visit at 30 months. We also found evidence for associations between specific genera of bacteria in infancy and negative affect. These results suggest associations between specific features of the gut microbiome and child behavior may differ based on timing of gut microbiome measurement.

Disruptive behavior increases the risk of developing more severe behavior problems later in life, including antisocial and criminal behavior. Parents behavior, and possibly their genetic makeup as well, plays a key role in shaping their children’s disruptive behavior. We examined gene-environment (parenting) correlations as underlying mechanisms for disruptive child behavior in a cross-sectional study. Polygenic scores for disruptive and externalizing behavior (PGS-DB and PGS-EXT) and parent-reported harsh and warm-supportive parenting were measured in 288 Dutch parent-child pairs (Child Mage = 6.26, SD = 1.31, 48% girls) with above-average parent-reported disruptive behavior. Harsh and warm-supportive parenting and children’s PGS-DB were associated with disruptive child behavior (β = .23, .10 and .15, respectively), but no evidence emerged for gene-environment correlations or genetic nurture. However, harsh parenting was found to partially mediate the link between parental PGS-EXT and disruptive child behavior (β = .04). These findings suggest that the choice of polygenic scores may influence the ability to detect genetic nurture as a relevant mechanism underlying disruptive child behavior.

Eating disorders (EDs) are particularly prevalent among young adult females. Previous research has shown that childhood trauma and reduced mentalizing abilities are involved in ED symptoms. The current study was aimed at testing the mediating effects of failures in mentalizing on the relationship between childhood trauma and ED risk among young adult females. The sample consisted of 409 Caucasian young adult females, aged between 18 and 30 years old (M = 23.45, SD = 2.76). The reported mean body mass index was within the normal range (M = 22.62; SD = 4.35). Self-report instruments were administered to assess the variables of interest. Structural equation modeling revealed that childhood trauma predicted increased failures in mentalizing (β = .36) and ED risk (β = .30), that failures in mentalizing predicted an increased ED risk (β = .35), and that the positive association between childhood trauma and ED risk was partially mediated by failures in mentalizing (indirect effect: β = .13). These findings suggest that ED symptoms might result from unprocessed and painful feelings embedded in child abuse and neglect. Clinical interventions focused at improving mentalizing abilities might reduce the ED risk among young adult females who have been exposed to childhood trauma.

Holistic frameworks of mental health outline that a focus on psychopathology does not represent an optimal approach to defining, measuring and treating mental health. Rather, theoretical, empirical, and applied psychological efforts should incorporate psychological well-being (PWB). Studies of PWB have overwhelmingly focused on adult populations, rendering a translation down to adolescence difficult. The current study explores the between-person, as well as within-person short-term, prospective relations between psychopathology and wellbeing within a community sample of adolescents (i.e., 553 youth aged 12 – 18, mean age: 14.97 years, 51.2% Male, 40.7% of participants identified as Hispanic (225 individuals), 38.5% identified as White (213 individuals), and 35.6% identified as Black (197 individuals), 3-wave, 1-year survey). Results demonstrated significant, negative between-person relations between psychopathology and PWB (bPHQ = −0.25, SE = 0.11, p = 0.021, bVDS = −0.39, SE = 0.15, p = 0.011). At the within-person level, consistent positive prospective relations were identified for violent-delinquent behaviors and PWB, such that increases in individual levels of violent-delinquent behaviors tended to forecast higher levels of PWB at the next follow-up (bPWBW2 = 0.21, SEPWBW2 = 0.076, p < 0.01; bPWBW3 = 0.14, SEPWBW3 = 0.051, p < 0.01). At the within-person level, prospective relations between depressive and PWB were not identified. Gender and racial/ethnic identities did not moderate findings.

Previous studies have suggested that nature contact is a protective factor for problem behavior in children. However, there remains a significant gap in research exploring the reciprocal relationship between nature contact and children’s problem behavior, as well as the underlying mechanisms driving this relationship. This study employed a longitudinal three-wave design involving 516 children in China (268 girls, Mage = 10.88 ± 0.66 years old at Time 3). Cross-lagged analyses indicated that nature contact and problem behavior negatively predicted each other over time, and prosocial behavior bidirectionally mediated the relationship between nature contact and problem behavior. These results provided evidence for the relationships among nature interaction, social development, and behavioral development in children. These findings suggested that promoting prosocial behavior could reduce problem behavior and enhance nature engagement, potentially serving as a strategy to foster comprehensive development in children.

This study examined associations between paternal, maternal, and dual-parental attention deficit hyperactivity disorder (ADHD) symptoms and child socioemotional functioning over the first two years of life, combined and separated by child sex. The sample included mothers (N = 3,207) and fathers (N = 3,211) from a prospective cohort in Canada. Parents completed the Adult ADHD Self-Report Scale within two weeks of childbirth. Children’s socioemotional functioning was assessed using the ASQ-SE at 6 months and the BITSEA at 12, 18, and 24 months. Paternal and maternal ADHD symptoms were associated with problems in child socioemotional development in the first two years of life, with significant differences based on parent and child sex. Paternal ADHD symptoms were associated with more socioemotional difficulties in boys (aOR 1.68, 95% CI 1.13–2.51) and fewer socioemotional difficulties in girls, while maternal ADHD symptoms were associated with more socioemotional problems in girls (aOR 2.09, 95% CI 1.24–3.52) and the entire sample, including both boys and girls, between 12 and 24 months. Dual-parental ADHD symptoms had the largest effect on socioemotional development (OR 4.43, 95% CI 1.14–17.16). Our findings provide evidence that exposure to paternal and maternal ADHD symptoms, especially when both parents exhibit symptoms, is associated with worse socioemotional outcomes during early childhood.

Narcissism is a personality trait characterized by a sense of being more important and entitled than others. Narcissism is high in adolescence and puts adolescents at risk of psychopathology and problematic social relationships. Why is narcissism persistent in adolescence? Bridging insights from developmental, clinical, social, and personality psychology, we examined whether adolescents (ages 11–15) high in narcissism maintain narcissism through downward social comparisons (e.g., “I am better than my classmates”), not downward temporal comparisons (e.g., “I am better now than when I was younger”). A cross-sectional study (N = 382, 97% Dutch) showed that adolescents higher in narcissism made more downward social and temporal comparisons. In a longitudinal study (N = 389, 99% Dutch), we assessed adolescents’ narcissism levels at the beginning of the school year and at 3-month follow-up. In-between, we captured adolescents’ comparisons through daily diary assessments. Adolescents higher in narcissism made more downward social and temporal comparisons. Downward social – but not temporal – comparisons partially mediated the 3-month stability of narcissism. In both studies, self-esteem was unrelated to downward comparisons. Thus, downward social – but not temporal – comparisons contribute to the maintenance of adolescent narcissism, and these comparisons constitute a potentially malleable developmental mechanism to curtail narcissism.

This study investigates the caregiver–child relationship as a proximal risk factor in the transactional development of youth personality pathology. 129 girls (aged 11–13 years), two-thirds of whom were oversampled for shy and fearful temperament, and their primary caregiver, participated in laboratory-based conflictual interactions. Trained observers rated positive and negative escalation, mutuality, relationship quality, and satisfaction. Concurrently and two years later, girls’ maladaptive traits were assessed via self- and caregiver-reports based on the DSM-5 Alternative Model for Personality Disorders (AMPD) domains (negative affect, detachment, antagonism, disinhibition, and psychoticism). Using a series of path models, we assessed whether dyadic interactions predicted changes in maladaptive traits. In dyads exhibiting reduced positive interaction patterns, specifically a lack of mutuality and relationship satisfaction, we observed increases in girls’ negative affect, detachment, disinhibition, and psychoticism. These patterns were more pronounced in girls’ self-reports. Negative escalation predicted girl- and caregiver-rated increases in antagonism. The study illustrates the importance of the caregiver–child relationship in the etiology of developmental personality pathology by establishing a link between observed caregiver–adolescent interactions and prospective changes in key domains of maladaptive traits. It expands the literature on dyadic interaction and developmental personality pathology to the dimensional framework of the AMPD.

Childhood maltreatment is a key risk factor for conduct disorder (CD), and the “ecophenotype hypothesis” suggests that maltreatment-related versus non-maltreatment-related CD are neurobiologically distinct. This may explain inconsistent findings in previous structural connectivity studies of CD. We tested this hypothesis by comparing youth with CD with (CD/+) versus without (CD/−) childhood physical or sexual abuse in white-matter microstructure. Diffusion tensor imaging data were collected from 100 CD and 169 control participants aged 9–18 years. Using Tract-Based Spatial Statistics, we compared the CD and control groups in fractional anisotropy, and axial, radial and mean diffusivity, then compared the CD/+ (n = 39) and CD/− (n = 61) subgroups and controls. The combined CD group had higher fractional anisotropy in the corpus callosum than controls. When divided by abuse history, only the CD/− subgroup exhibited higher corpus callosum fractional anisotropy than controls; the CD/+ subgroup did not differ from controls. Comparing the CD subgroups, the CD/+ subgroup displayed higher superior longitudinal fasciculus axial diffusivity than the CD/− subgroup. Notably, sex-stratified analyses yielded different findings in all-male and all-female samples. Findings support the ecophenotype hypothesis, demonstrating microstructural differences between the CD/+ and CD/− subgroups and emphasizing the importance of considering abuse/maltreatment (and sex) in future studies.

Adolescents who experience bereavement following suicide are at increased risk for adverse outcomes, including depression. However, there is limited research on the heterogeneity of depressive symptoms or its long-term course among this population. Using a self-reported 3-item version of the Center for Epidemiologic Studies Depression Scale (CES-D) administered across five waves spanning from adolescence to adulthood (1994–2018, with intervals of 1, 5, 7, and 9 years), we identified trajectories of depressive symptoms over a 24-year span in a sample of adolescents (n = 236) who reported at baseline having lost a family member or friend to suicide in the last 12 months. We identified three distinct depressive symptom trajectories: Stable low symptoms (77.5%), initially high but gradually declining symptoms (16.9%), and initially low but gradually increasing symptoms (5.5%). Race, neuroticism, sleep quality, and age were significant predictors that differentiated membership among the three trajectory groups. Implications for developing personalized assessment and intervention are discussed.

Empirical tests of the “cycle of maltreatment” hypothesis have typically focused on the presence or absence of child maltreatment across generations. However, this narrow focus does not account for diverse intergenerational pathways of maltreatment. This systematic review and meta-analysis synthesizes data to determine the distribution of cycle maintainers, breakers, initiators, and unaffected families (i.e., controls). Of the 65 independent studies (80 samples), 30 examined intergenerational cycles of maltreatment broadly, while 27 reported data for physical abuse, 17 sexual abuse, 5 neglect, and 1 emotional abuse specifically. For maltreatment, 17.1% (95%CI: 12.1%, 22.1%) were cycle maintainers, 23.6% (95%CI: 18.0%, 29.2%) were cycle breakers, 11.4% (95%CI: 7.8%, 15.1%) were cycle initiators and 47.8% (95%CI: 39.7%, 55.9%) controls. Thus, although a parent’s maltreatment history is a risk factor, results suggest that a greater proportion of parents break the cycle of maltreatment versus maintain it. Moderator analyses showed that study design, assessment methods, and demographic characteristics influence maltreatment transmission rates. Intergenerational patterns of physical, sexual, and emotional abuse and neglect are also detailed. Our findings underscore the complexity of intergenerational maltreatment, highlighting the need to explore not only its maintenance but also the protective factors that help break cycles and the risk factors that drive its initiation.

While there is evidence that childhood maltreatment (CM) is positively associated with drug use (DU), the strength and difference of the association between CM and its subtypes (hereafter CM + ST) and DU remains to be further explored. A multilevel meta-analysis was conducted on 101 independent studies reporting 333 effect sizes (N = 132,341; Mage = 24.65; 43.80%males). Results showed significantly positive correlations between CM + ST and DU (range from 0.109 to 0.185). The results of the subgroup analysis revealed notable disparities in the correlations between distinct CM subtypes and DU (F = 5.358, P<0.01). Specifically, the effect size for childhood sexual abuse (CSA) was significantly lower than childhood emotional maltreatment (CEM) and childhood physical maltreatment (CPM), while no significant difference was noted between the CEM and CPM groups. These effect sizes also varied across regions, drug types, gender, detection rate of CM, the presence or absence of alcohol in substances, publication status and measurement method. The significant yet differing correlations between different subtypes of CM and DU to some extent support the principle of equality in psychopathology. These findings help explain the relationship between CM + ST and DU laying the groundwork for further research into the intricate and complex associations between CM and DU.

Physical and sexual abuse have far-reaching mental and behavioral health consequences, extending across the lifespan and, in some cases, across generations. However, empirical work in this area is limited by cross-sectional study designs, short follow-up durations, and data analytic techniques that fail to capture the nuanced developmental processes through which caregivers and children impact one another. The present study investigated the cross-lagged and bidirectional pathways between maternal childhood victimization, depression, harsh parenting, and their children’s externalizing symptoms over a 10-year period. Participants were 818 mother-child dyads prospectively identified as at-risk for family violence when children were four years old. Traditional cross-lagged panel modeling (CLPM) and random-intercept cross-lagged panel modeling (RI-CLPM) documented that maternal depression, harsh parenting, and child externalizing problems — all predicted by mothers’ early abuse experiences — exacerbated one another across time. Discrepancies between the CLPM and RI-CLPM highlighted the advantages, disadvantages, and methodological implications of each approach. Findings highlight maternal psychopathology and parenting as key mechanisms in the intergenerational impact of abuse, emphasizing the importance of trauma-informed, parent-mediated interventions for breaking long-term cycles of family dysfunction. The present findings support separating out between-person, trait-like components when interpreting cross-lagged associations, as these may confound within-person effects.

The development of parenting behaviors is influenced by the coparenting relationship – how parents coordinate and manage their shared parenting responsibilities. However, mothers and fathers may exhibit varying degrees of susceptibility to the coparenting relationship, and different factors may further shape their susceptibility. Longitudinal data from 182 different-sex dual-earner parent families were used (86% White, mostly middle- and upper-class families). Observations of coparenting and parenting behaviors were obtained at 3 and 9 months postpartum. Mothers and fathers reported their beliefs about parental roles and parenting at the third trimester of pregnancy and reported their parenting self-efficacy and infant temperament at 3 months. The results of cross-lagged panel models showed that the associations between coparenting and parenting were not significantly different for mothers and fathers. However, the moderators that shaped the associations between coparenting and parenting were distinct for mothers and fathers. Fathers’ less progressive parental role beliefs strengthened the association between supportive coparenting and positive parenting, and mothers’ lower parenting self-efficacy and children’s lower regulatory capacity enhanced the link between undermining coparenting and negative parenting. Implications for research and practice are discussed.

The COVID-19 pandemic disrupted the population’s lives. Stressful conditions during the lockdown and the reintroduction to a changed social environment emotionally affected children and adolescents. The aim of this work was to study anxiety and depressive symptoms in Italian, Spanish, and Portuguese children and adolescents aged 3 to 18 years at different moments of the COVID-19 pandemic: April 2020 (during confinement), September 2020 (with the schools’ reopening), and September 2023 (with the situation restored). Parents of 1,097 children participated in at least one assessment, completing measures of child emotional symptoms online. Cases with subclinical symptoms of anxiety and depression were higher compared to pre-pandemic studies. Overall, anxiety increased from April 2020 to September 2020, decreasing in September 2023 with no differences compared to the first assessment. Depression was high in April 2020 but decreased in September 2020, with no significant differences three years later, in September 2023. Cross-country comparisons at each point are discussed. Moreover, boys showed higher levels of depression during the pandemic compared to girls. Older children, compared to younger ones, had more anxiety and depressive symptoms throughout all the moments. These findings highlight the emotional impact of the pandemic and its conditions on children and adolescents.